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>>>>> Help ME Circle <<<<
>>>> 16 July 2011 <<<<
Editorship : j.van.roijen@chello.nl
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The authors below from London are speaking about:
*CFS as defined by recent criteria*
The only recent criteria, I can think of are the
terrible *unwellness criteria* by Reeves, Jones,
Unger, Vernon et al.
The CDC estimated that six to ten times more
people have this illness than their previous reports in
the US.
These figures were received with cheer on the
website of the US patient charity CAA by the director
Kim McCleary, who get a salary, which is the same as
that of the Minister President of The Netherlands.
Or do they mean the fraudulent Nice Guidelines from
London?
I'm too sick and brainfogged to think clearly, so
forgive me and blame it on ME, but one thing is for
sure, these scientists come with very old news.
~jan van roijen
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http://www.ncbi.nlm.nih.gov/pubmed/21756995
Brain Behav Immun. 2011 Jul 2. [Epub ahead of print]
Chronic fatigue syndrome, the
immune system and viral infection.
Bansal AS, Bradley AS, Bishop KN, Kiani S, Ford B.
Dept. of Immunology, Epsom and St. Helier University
Hospitals NHS Trust, Carshalton, Surrey, SM5 1AA and
Chronic Illness Research Team, Stratford Campus,
University of East London, London E15 4LZ, UK.
Abstract
The chronic fatigue syndrome (CFS), as defined by
recent criteria, is a heterogeneous disorder with a
common set of symptoms that often either follows a
viral infection or a period of stress.
Despite many years of intense investigation there is
little consensus on the presence, nature and degree
of immune dysfunction in this condition.
However, slightly increased parameters of
inflammation and pro-inflammatory cytokines such as
interleukin (IL) 1, IL6 and tumour necrosis factor
(TNF) alpha are likely present.
Additionally, impaired natural killer cell function
appears evident.
Alterations in T cell numbers have been described by
some and not others.
While the prevalence of positive serology for the
common herpes viruses appears no different from
healthy controls, there is some evidence of viral
persistence and inadequate containment of viral
replication.
The ability of certain herpes viruses to impair the
development of T cell memory may explain this viral
persistence and the continuation of symptoms.
New therapies based on this understanding are more
likely to produce benefit than current methods.
Copyright =A9 2011. Published by Elsevier Inc.
PMID: 21756995 [PubMed - as supplied by publisher]
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